Researchers at the Medical University of Vienna and the Medical University of Innsbruck discovered that SARS-CoV-2 hijacks ...

Hijacking host proteins for effective complement resistance By propagating SARS-CoV-2 in human cells the researchers discovered that the virus particles acquire the cellular proteins CD55 and CD59.

Researchers at the Medical University of Vienna and the Medical University of Innsbruck discovered that SARS-CoV-2 hijacks three important host proteins that dampen the activity of the complement ...

Binding activity by ELISA assay against SARS-CoV-2 variants: The 40592-MM117 clone binds to spike proteins from the Omicron variant. It also recognizes the wild type and the Delta variants.

Over the last few years, the SARS-CoV-2 virus, responsible for COVID-19, has undergone significant changes, evolving from the ...

Structural proteins--N, M and E--move inside the cell, where they help new virions form. A SARS-CoV-2 particle enters a person's nose or mouth and floats in the airway until it brushes against a ...

A recent study suggests that lingering SARS-CoV-2 proteins in the blood of long COVID sufferers could be a key factor in their ongoing symptoms. This discovery offers new insights into the complex ...

In a recent paper, Henry’s team detailed the aeration and feeding strategies they used as they scaled production of SARS-CoV-2 spike protein from a 0.75 L bioreactor to a 1 L multisystem bioreactor.

Researchers at the Medical University of Vienna and the Medical University of Innsbruck discovered that SARS-CoV-2 hijacks three important host proteins that dampen the activity of the complement ...